Watch more clips of Dr. James Kelly - https://www.youtube.com/playli....st?list=PLe2Je5-cHxP And for more information about brain injury and PTSD, please visit us at https://www.brainline.org.

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Curious about LASIK eye surgery? NVISION's Dr. Richard Mauer talks risks, life-changing benefits, and outcomes (plus why he loves what he does!).

Want to start your journey to better vision? Schedule your complimentary consult today! https://bit.ly/3H2i0FU

NVISION: The Eye Doctors' #1 Choice in LASIK and Laser Cataract Surgery

The dentin is a hard tissue that forms the bulk of the tooth. It is similar to bone but is slightly harder, although softer than enamel. The dentin has numerous dentinal tubules that run across its length. Each dentinal tubule houses the cytoplasmic process of an odontoblast (odontoblastic process).
📄Notes for the video: https://www.hackdentistry.com/....bundles/revision-nin

💻Website: https://www.hackdentistry.com/
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Study resources on our website-
📖Oral pathology Revision Ninja (Notes, Videos & MCQs): https://www.hackdentistry.com/bundles/oral-pathology-revision-ninja
📖Oral Histology Revision Ninja (Notes, Videos & MCQs): https://www.hackdentistry.com/....bundles/revision-nin
📖Periodontics Revision Ninja (Notes & MCQs): https://www.hackdentistry.com/bundles/perio-rn
📖Question Bank: https://www.hackdentistry.com/bundles/question-bank
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References and further reading:
💡Berkovitz BKB, Hollan GR, Moxham BJ. Oral Anatomy, Histology and Embryology. 4th ed. Mosby Elsevier; 2009.
💡Nanci A. Tencate’s Oral Histology. Development, Structure and Function. 8th ed. Elsevier; 2013.
💡Kumar GS. Orban’s Oral Histology and Embryology.13th ed. Elsevier; 2011.
💡Avery JK. Oral development and Histology. 3rd ed. Thieme Medical Publishers; 2002.

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Dimples creation Surgery/ Dimpleplasty is a type of plastic surgery used to create dimples on the cheeks. Dimples are the indentations that occur when some people smile. They’re most often located on the bottoms of the cheeks. Not everyone is born with this facial trait. In some people, dimples are naturally occurring due to defect in smiling muscle. Dimples are perceived as a sign of beauty.

How dimples are formed ?
Dimples are formed because of the defect in the smiling muscle called as Zygomaticus Major. A very small part of this muscle is attached to skin. So whenever a person smiles the skin lying over this defect gets wedged into it, in the connective tissue, creating dimples in the cheeks which is perceived as a beautiful features on one's face.

Procedure of Dimple Creation
1.Before the surgery the doctors mark the position of the dimple based on patients on where the patient wants the dimple. The doctor also guides the patient regarding the position of dimples.
2.This surgery is performed under local anaesthesia .
3.Doctors perform this surgery from inside the mouth (inside of cheek) in order ensure there are no scars on the face.A small cut of 3-4mm is made inside the cheek area.
4.After making this incision, depending on where you want the dimple, a suture is passed through. This connects your skin to the underlying muscle, creating the dimple. You can see the dimple immediately.

Static and Dynamic Dimple
This type of dimple is called as static dimple.These dimples do not change in position or depth with animation. They remain like a sore thumb even when the person has no expression. As an absorbable stitch is used used during surgery, gradually the skin will, however, flatten out when the stitch is absorbed. The internal scarring —which takes place — will connect the muscle to the skin, creating a dimple which becomes prominent when one smiles and the static dimple will convert into dynamic dimples after two months .

Dimple creation surgery is a daycare procedure. It only takes 30 minutes, and you can go home soon after surgery. You are fit to resume your daily activities from the next day itself.

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Perineal rectosigmoidectomy

Transoral Access in Endoscopic Thyroid Surgery Background: The number of patients demanding endoscopic neck surgery is rising. The access trauma of the axillary, breast and chest approaches is bigger than in open or video assisted surgery. We tested the feasibility of he sublingual transoral access which is in our opinion the only real minimally...-invasive extracollar endoscopic access to the thyroid gland Methods: We performed an experimental investigation in a porcine model. In 10 pigs we made 10 endoscopic transoral thyroidectomys with a modified axilloscope with the help of ultrasonic scissors and a neuro-monitoring system for identification of the recurrent laryngeal nerve. Results: The average operation time from the introduction to the removal of the obturator just above the larynx was 57 seconds. The mean operation time was 43 minutes. With the help of the neuro-monitoring system we proved in all cases the function of the recurrent laryngeal nerve on both sides. The pigs were observed for another two hours after operation. During and after the operation no complications appeared. Conclusions: We could show that the endoscopic transoral thyroid resection in pigs is possible and save. Our results might be useful for using this access for endoscopic thyroid resection in humans.

This is the CT of a 43 year old male patiënt with infected necrotizing pancreatitis that will undergo a VARD procedure; Videoscopic Assisted Retroperitoneal Debridment. Two weeks before this procedure two large bore percutaneous drains were placed in the peripancreatic collection. The patient i...s placed in supine position with the left side 30 degrees elevated. A 5-7 cm subcostal incision is made in the left flank. With help of CT images and by following the percutaneous drain, the subcutaneous tissue and the fascia are dissected and we enter the retroperitoneal peripancreatic collection. First, with a regular suction device any pus encountered is removed. Two long sympathectomy hooks are inserted in order to keep in the incision open. We than insert the zero degree laparoscope. The first necrosis encountered is removed under direct sight with the use of long grasping forceps. Following the percutaneous drain deeper into the cavity, parts of loosely adherent necrotic material are removed. Gently pulling we remove the necrotic tissue. The suction device is helpful in removing any fluid obstructing the view. Complete necrosectomy is not the ultimate aim of this procedure. Only loosely adherent pieces of necrosis are removed thereby keeping the risk of tearing underlying blood vessels to a minimum. In the rare case of extensive bleeding, the retroperitoneal cavity can be easily packed, either awaiting the bleeding to definitely stop or to act as a bridge to angiographic coiling. This patient is now 6 weeks after onset of disease. We always try to postpone surgical intervention, if possible up to 30 days. On the left side of the collection is the percutaneous drain. In this patient the drain had worked well for 2 weeks. When the patient deteriorated again it was decided to perform the VARD procedure. Large pieces of necrotic pancreas can be removed with VARD. This is a big advantage ov VARD over pure endosopic or percutaneous techniques. When all the necrotic tissue is removed we clean the cavity. Two drains are left in situ as a postoperative lavage system. The VARD procedure is performed via a 6 cm incision, which is closed and continuous postoperative lavage started immediately.

Hysteroscopy adhesiolysis for treating Ashermann syndrome

A video showing how to draw blood for sampling

foot cast used in Toe Fractures

Bat Ears Correction Plastic Surgery

Amyotrophic lateral sclerosis The disorder causes muscle weakness and atrophy throughout the body caused by degeneration of the upper and lower motor neurons. Unable to function, the muscles weaken and atrophy. Affected individuals may ultimately lose the ability to initiate and control all voluntary movement, although bladder and bowel sphincters and the muscles responsible for eye movement are usually, but not always, spared. Cognitive function is generally spared for most patients although some (~5%) also have frontotemporal dementia.A higher proportion of patients (~30-50%) also have more subtle cognitive changes which may go unnoticed but are revealed by detailed neuropsychological testing. Sensory nerves and the autonomic nervous system, which controls functions like sweating, are generally unaffected but may be involved for some patients. The earliest symptoms of ALS are typically obvious weakness and/or muscle atrophy. Other presenting symptoms include muscle fasciculation (twitching), cramping, or stiffness of affected muscles; muscle weakness affecting an arm or a leg; and/or slurred and nasal speech. The parts of the body affected by early symptoms of ALS depend on which motor neurons in the body are damaged first. About 75% of people contracting the disease experience "limb onset" ALS i.e. first symptoms in the arms ("upper limb", not to be confused with "upper motor neuron") or legs ("lower limb", not to be confused with "lower motor neuron"). Patients with the leg onset form may experience awkwardness when walking or running or notice that they are tripping or stumbling, often with a "dropped foot" which drags along the ground. Arm-onset patients may experience difficulty with tasks requiring manual dexterity such as buttoning a shirt, writing, or turning a key in a lock. Occasionally, the symptoms remain confined to one limb for a long period of time or for the whole course of the illness; this is known as monomelic amyotrophy. About 25% of cases are "bulbar onset" ALS. These patients first notice difficulty speaking clearly or swallowing. Speech may become slurred, nasal in character, or quieter. Other symptoms include difficulty swallowing, and loss of tongue mobility. A smaller proportion of patients experience "respiratory onset" ALS where the intercostal muscles that support breathing are affected first. Regardless of the part of the body first affected by the disease, muscle weakness and atrophy spread to other parts of the body as the disease progresses. Patients experience increasing difficulty moving, swallowing (dysphagia), and speaking or forming words (dysarthria). Symptoms of upper motor neuron involvement include tight and stiff muscles (spasticity) and exaggerated reflexes (hyperreflexia) including an overactive gag reflex. An abnormal reflex commonly called Babinski's sign (the big toe extends upward and other toes spread out) also indicates upper motor neuron damage. Symptoms of lower motor neuron degeneration include muscle weakness and atrophy, muscle cramps, and fleeting twitches of muscles that can be seen under the skin (fasciculations). Around 15–45% of patients experience pseudobulbar affect, also known as "emotional lability", which consists of uncontrollable laughter, crying or smiling, attributable to degeneration of bulbar upper motor neurons resulting in exaggeration of motor expressions of emotion.

This video goes through a case study of a 40 year old patient getting breast reduction surgery in NYC by board certified plastic surgeon Carlin Vickery of 5th Avenue Surgery located in New York, NY. For more information on this procedure please call our office at (212) 288-9800. This content is intended for general information only and does not replace the need for personal advice from a qualified health professional.

Replacement of the wrist joint with an artificial wrist joint is often needed when a traumatic injuries or severe degenerative affecting the wrist (osteoarthritis and rheumatoid arthritis) occur.

A very funny song made by the staff of the Ob/Gyn Gangnam style

Inserting main port in laparoscopy