Situs inversus is a condition in which the arrangement of the internal organs is a mirror image of normal anatomy. It can occur alone (isolated, with no other abnormalities or conditions) or it can occur as part of a syndrome with various other defects. Congenital heart defects are present in about 5-10% of affected people. The underlying cause and genetics of situs inversus are complex. Familial cases have been reported.[1][2]

Transjugular intrahepatic portosystemic shunt or transjugular intrahepatic portosystemic stent shunting (commonly abbreviated as TIPS or TIPSS) is an artificial channel within the liver that establishes communication between the inflow portal vein and the outflow hepatic vein.

The accumulation of ascitic fluid represents a state of total-body sodium and water excess, but the event that initiates the unbalance is unclear. Although many pathogenic processes have been implicated in the development of abdominal ascites, about 75% likely occur as a result of portal hypertension in the setting of liver cirrhosis, with the remainder due to infective, inflammatory, and infiltrative conditions. Three theories of ascites formation have been proposed: underfilling, overflow, and peripheral arterial vasodilation. The underfilling theory suggests that the primary abnormality is inappropriate sequestration of fluid within the splanchnic vascular bed due to portal hypertension and a consequent decrease in effective circulating blood volume. This activates the plasma renin, aldosterone, and sympathetic nervous system, resulting in renal sodium and water retention. The overflow theory suggests that the primary abnormality is inappropriate renal retention of sodium and water in the absence of volume depletion. This theory was developed in accordance with the observation that patients with cirrhosis have intravascular hypervolemia rather than hypovolemia. The most recent theory, the peripheral arterial vasodilation hypothesis, includes components of both of the other theories. It suggests that portal hypertension leads to vasodilation, which causes decreased effective arterial blood volume. As the natural history of the disease progresses, neurohumoral excitation increases, more renal sodium is retained, and plasma volume expands. This leads to overflow of fluid into the peritoneal cavity. The vasodilation theory proposes that underfilling is operative early and overflow is operative late in the natural history of cirrhosis. Although the sequence of events that occurs between the development of portal hypertension and renal sodium retention is not entirely clear, portal hypertension apparently leads to an increase in nitric oxide levels. Nitric oxide mediates splanchnic and peripheral vasodilation. Hepatic artery nitric oxide synthase activity is greater in patients with ascites than in those without ascites. Regardless of the initiating event, a number of factors contribute to the accumulation of fluid in the abdominal cavity. Elevated levels of epinephrine and norepinephrine are well-documented factors. Hypoalbuminemia and reduced plasma oncotic pressure favor the extravasation of fluid from the plasma to the peritoneal fluid, and, thus, ascites is infrequent in patients with cirrhosis unless both portal hypertension and hypoalbuminemia are present.

How to examine a patient's wrist if you suspect a scaphoid fracture: by Cape Town Emergency Medicine

today we talk about Amniotic fluid during your pregnancy! Looking forward to your comments.

Very small currents can be imperceptible. Larger current passing through the body may make it impossible for a shock victim to let go of an energized object. Still larger currents can cause fibrillation of the heart and damage to tissues. Death caused by an electric shock is called electrocution.

Fracture of meta-diaphyseal junction of the fifth metatarsal of the foot

Abortion pills can help one to terminate pregnancy at home without letting others know- which is very true. But if you are not cautions enough, it wouldn’t be secured, doing in private. This might be due to the nosy neighbor or friends who peep up any time in the day. Arrange things for and take precautions, to avoid getting your private affair publicly few seconds.

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Skin Whitening Tips, Vitamin C For Skin Whitening, Skin Whitening Before And After, Skin Whitening. http://skin-whitening.good-info.co Each and every person wants a clean and radiant skin. Some spend fortunes on cosmetic products that will lighten the skin and remove all the imperfections, others spend their money on esthetic operations in the hope that their skin will look perfect. Lastly, there are people trying to fake a healthy skin by using all kinds of makeup that will cover the imperfections and leave the impression that the skin is healthy and has no scars, wrinkles or spots. All these people are looking for a way through which they can make their skin look good. Yet, what they have not taken into consideration is the power of natural ingredients. Fruits, vegetables and products coming from animals are great sources of anti-oxidants and are rich in substances which can whiten the skin, moisturize it, attenuate the fine lines and wrinkles and give it elasticity. There are many natural ingredients which can be used in order to remove the dark spots and whiten the skin and as many reasons to start trying them. The first reason for which you should try the natural skin whitening ingredients is the fact that they have no side effects. Because most of the ingredients with which the homemade skin whitening recipes are made are natural, and are used in our everyday diet, the human body tolerates them very well and responds positively to the ingredients they contain. In addition to that, because you know what you put in that homemade recipe, you know if your skin will going to react negatively to it. There are certain products (fruits, vegetables, animal products) to which certain people are allergic. Exclude those ingredients from your recipes and you know you will obtain a 100% safe homemade product. Click Here. http://skin-whitening.good-info.co

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http://eliminar-celulite.plus101.com --- Eliminar Celulite, O Que Fazer Para Acabar Com A Celulite, Como Tirar Celulite Das Pernas. Mas as razões que vou compartilhar são diferentes das que a maioria das outras fontes está tentando fazê-la acreditar. Há um mito fazendo com que algumas mulheres acreditem que certos alimentos e nutrientes irão “eliminar as toxinas que estão causando a celulite”. ISSO É TOTALMENTE FALSO, porque não há toxinas em ou sob sua pele. Se houvesse toxinas se acumulando e ficando presas sob sua pele, você estaria morta. Simples assim. Nosso corpo foi feito para remover toxinas com muita eficácia. Este processo fisiológico acontece 24 horas por dia, 7 dias por semana, sem parar, o tempo todo. Então, a ideia não comprovada de que “toxinas” são a causa de sua celulite significa que a celulite não pode ser revertida ao “eliminá-las” com alguns alimentos, porque elas não estão lá, para começar. Mas não se preocupe, porque eis o que o planejamento alimentar apropriado pode fazer para reverter, ou prevenir, a raiz da causa da celulite em suas pernas, bumbum, quadris e coxas. Uma verdadeira dieta contra a celulite fornece nutrientes em quantidades que impactam positivamente a regulagem e equilíbrio dos hormônios femininos. Esta é a razão principal de o Planejamento Alimentar/Dieta Contra Celulite do "Adeus Celulite" só estar disponível para mulheres que começam com o Método de Exercícios SYMULAST do programa Adeus Celulite. Então se você estiver interessada, vá para: http://eliminar-celulite.plus101.com

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Hemorrhoids (HEM-uh-roids), also called piles, are swollen veins in your anus and lower rectum, similar to varicose veins. Hemorrhoids have a number of causes, although often the cause is unknown. They may result from straining during bowel movements or from the increased pressure on these veins during pregnancy.

Cartilage is a slippery tissue that provides a smooth surface for joint motion and acts as a cushion between the bones. Synovium is soft, and it lines the joints. It produces fluid, called synovial fluid, for lubrication, and it supplies nutrients and oxygen to the cartilage. As these functions break down, they no longer protect the bones of the knee joint, and bone damage occurs. OA of the knee can cause pain and stiffness. The symptoms worsen over time

After Sammyra’s knee injury, Marvin Smith, MD, orthopaedic surgeon at Memorial Sports Medicine Center, helped her get back on the volleyball court and playing pain free. Following a thorough examination, meniscus surgery and rehabilitation got Sammyra back to playing with her college team within two months. Learn more about how Memorial Sports Medicine Center helps athletes move forward at MHS.net/SportsMedicine.
To learn more about Dr. Smith, visit his physician profile page at: https://www.mhs.net/physicians/s/smith-marvin-k

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A complete organized library of all my videos, digital slides, pics, & sample pathology reports is available here: https://kikoxp.com/posts/5084 (dermpath) & https://kikoxp.com/posts/5083 (bone/soft tissue sarcoma pathology)

Topics discussed:

Epidermis:
Layers of epidermis: 0:10
Melanocytes vs Keratinocytes: 5:16
Langerhans cells: 10:10 & 33:30 & 57:30

Dermis:
Papillary and reticular dermis: 11:50
Three types of white empty spaces on a slide: vessels, glands/ducts/cysts, or artifact: 15:25
Blood vessels & nerves: 18:24 & 48:50 & 58:59
Arrector pili & other dermal smooth muscle: 20:00

Adnexal:
Sebaceous gland: 21:10
Hair follicle 23:14
Eccrine sweat glands and ducts 24:45 & 50:00
Gland/duct vs blood vessel 27:20 & 48:50
Apocrine glands: this video https://kikoxp.com/posts/7837 (at 12:30)
Acrosyringium: this video https://kikoxp.com/posts/7837 (at 10:00)

Three types of pink bundles: smooth muscle, nerve, dense connective tissue: 27:50

Acral skin (palm sole) with contact dermatitis 29:37
Parakeratosis 30:00
Perivascular lymphocytes 30:40
Eosinophils vs neutrophils 31:20
Spongiosis with desmosome keratinocyte spines 32:10
Spongiotic vesicles with Langerhans cells 33:30
Normal acral skin (palm & sole) with stratum lucidum 34:20
Normal glomus body/apparatus (canal of Sucquet-Hoyer) 35:40
Nerve 36:46 & 51:50
Adipose tissue (white fat cells) in subcutis with Lochkern 37:55
Normal scalp skin with large anagen hair follicles: 39:30
Hair follicle anatomy (bulb/matrix, inner root sheath, outer root sheath, hair shaft, isthmus, infundibulum): 40:55 (labeled images):
https://kikoxp.com/posts/3661 & https://kikoxp.com/posts/7899
Pacinian corpuscle 50:40
Meissner corpuscle 1:02:28

Dense regular connective tissue (Fascia/Tendon/Ligament) vs Smooth Muscle 53:00

Basic Normal Skin Immunohistochemistry:
-cytokeratin in epidermis: 55:33
-S100 in melanocytes and Langerhans cells and adipocytes: 57:30
-Desmin in smooth muscle (arrector pili and blood vessels): 58:59
-CD31 in endothelial cells of blood vessels: 59:33
-SOX-10 in melanocytes: 1:00:40

Digit/Finger/Toe histology (amputation for subungual acral melanoma) 1:04:10 & 1:08:30
-bone 1:05:40
-glomus body 1:05:15
-tendon/ligament 1:06:10
-artery 1:06:58
-fingernail/toenail 1:08:54
-acrosyringium 1:10:45

Solar elastosis (what wrinkles look like microscopically!) 1:11:50

Other videos you might like:
Tendon vs Nerve Histology Made Simple with the Ramen Noodle Sign (of Fulton) video: https://kikoxp.com/posts/4466
Melanocytes vs Keratinocytes made easy video: https://kikoxp.com/posts/3802
Blood Vessel vs Gland vs Artifact Made Easy video: https://kikoxp.com/posts/4808

The basic normal structures of the skin discussed and described by a dermatopathologist. This material is intended for use by medical students, junior pathology or dermatology residents, or for anyone else studying normal human histology. Special thanks to two of my medical students at UAMS for helping make this video possible. Miki Lindsey convinced me that I really needed to sit down and record this video. Akash Patel took time to edit the video and make it ready for YouTube. My sincere thanks to both of them for helping me overcome procrastination.

Huge thanks to Abigail Cline, a medical student at Medical College of Georgia, for volunteering to type a transcript of this ENTIRE video (over 14,000 words!) so that I could provide closed caption subtitles for those with hearing impairments and for those who may need assistance in understanding spoken English (particularly given how quickly I speak!). You can access a text version of her transcript of my video here: https://kikoxp.com/posts/5390

Correction - I made a mistake in the video. I said that sebaceous gland secretions are turned into smelly substances by bacteria and that this makes body odor. That is incorrect. That is actually true of APOCRINE gland secretions not sebaceous secretions.

Also, in the past I used "keratinocyte" and "squamous cell" interchangeably (this is because in dermatopathology, we see and talk about squamous cell carcinomas all the time, and those tumors are composed of keratinocytes). But technically, in normal skin histology, "squamous cell" refers only to the flattened keratinocytes in the superficial epidermis. Thankfully, a histology PhD colleague pointed this out to me and corrected my lazy nomenclature!

Please check out my Soft Tissue Pathology & Dermatopathology survival guide textbooks: http://bit.ly/2Te2haB ‬

This video is geared towards medical students, pathology or dermatology residents, or practicing pathologists or dermatologists. Of course, this video is for educational purposes only and is not formal medical advice or consultation.

Presented by Jerad M. Gardner, MD. Please subscribe to my channel to be notified of new pathology teaching videos.

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