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Have you heard any medical lingo you've thought is strange? Funny healthcare speaker Dr. Brad Nieder discusses funny medical terminology he's learned in his medical career. He brings his medical comedy to a healthcare conference, describing how he didn't know what "stat" meant.
He goes on about how he thought up many funny terms he could say in return to the doctor who introduced him to the word. His healthcare comedy makes the crowd burst with laughter.
Dr. Brad knows how to adapt his hilarious real-life stories into customized presentations for any in-person or virtual event. Watch more of his videos as a medical comedian and all-around funny guy by browsing his videos.
Experts once believed that atherosclerosis, or hardening of the arteries, developed when too much cholesterol clogged arteries with fatty deposits called plaques. ... Researchers have discovered how diabetes, by driving inflammation and slowing blood flow, dramatically accelerates atherosclerosis.Mar 17, 2008 Past studies had shown diabetes to worsen atherosclerosis, but its exact link to related inflammation had remained unclear. The current results provides the first mechanistic description of how diabetes takes away the ability of fast blood flow force to protect blood vessels, arguing that it does so by interfering with ERK5 and its signaling partners.
Tuberous breast deformity is a congenital breast anomaly that becomes manifest at the time of puberty and breast development. The three components of tubular deformity usually include, pseudoherniation of breast tissue into the nipple areolar complex, poorly defined inframammary fold and flattening of the lower pole of the breast which leads to a conical tubular shape. Stuart Linder M.D. 9675 BRIGHTON WAY, SUITE 420 BEVERLY HILLS CA 90210 (310) 275-4513
The 12-lead ECG is a vital tool for EMT’s and paramedics in both the prehospital and hospital setting. It is extremely important to know the exact placement of each electrode on the patient. Incorrect placement can lead to a false diagnosis of infarction or negative changes on the ECG.
Myelomeningocele remains the most complex congenital malformation of the central nervous system that is compatible with life. This lesion results when the neural tube fails to fold normally during postovulatory Days 21 to 27.[6] The exact cause of disorders remains under some historical debate and is not within the scope of this paper. Myelomeningocele within the context of this discussion refers only to lesions that involve an open caudal neural tube defect on the surface of the skin
What is the spleen and what causes an enlarged spleen (splenomegaly)? The spleen sits under your rib cage in the upper left part of your abdomen toward your back. It is an organ that is part of the lymph system and works as a drainage network that defends your body against infection. White blood cells produced in the spleen engulf bacteria, dead tissue, and foreign matter, removing them from the blood as blood passes through it. The spleen also maintains healthy red and white blood cells and platelets; platelets help your blood clot. The spleen filters blood, removing abnormal blood cells from the bloodstream. A spleen is normally about the size of your fist. A doctor usually can't feel it during an exam. But diseases can cause it to swell and become many times its normal size. Because the spleen is involved in many functions, many conditions may affect it.
What are diverticula? Diverticula are outpouchings that most commonly happen in the sigmoid colon of the large intestine. The presence of a diverticulum is defined as diverticulosis, whereas diverticulitis describes an inflamed diverticulum
Take your left leg and place your ankle against the knee. Hold the position for a moment before changing legs. This helps stretch the tiny piriformis muscle, which sometimes becomes inflamed and presses against the sciatic nerve causing pain. Repeat by switching sides and doing the same exercise with the other leg.
Bronchiectasis is an abnormal dilation of the proximal and medium-sized bronchi (>2 mm in diameter) caused by weakening or destruction of the muscular and elastic components of the bronchial walls. Affected areas may show a variety of changes, including transmural inflammation, edema, scarring, and ulceration, among other findings. Distal lung parenchyma may also be damaged secondary to persistent microbial infection and frequent postobstructive pneumonia. Bronchiectasis can be congenital but is most often acquired.[9] Congenital bronchiectasis usually affects infants and children. These cases result from developmental arrest of the bronchial tree. Acquired forms occur in adults and older children and require an infectious insult, impairment of drainage, airway obstruction, and/or a defect in host defense. The tissue is also damaged in part by the host response of neutrophilic proteases, inflammatory cytokines, nitric oxide, and oxygen radicals. This results in damage to the muscular and elastic components of the bronchial wall. Additionally, peribronchial alveolar tissue may be damaged, resulting in diffuse peribronchial fibrosis.[12] The result is abnormal bronchial dilatation with bronchial wall destruction and transmural inflammation. The most important functional finding of altered airway anatomy is severely impaired clearance of secretions from the bronchial tree. Impaired clearance of secretions causes colonization and infection with pathogenic organisms, contributing to the purulent expectoration commonly observed in patients with bronchiectasis. The result is further bronchial damage and a vicious cycle of bronchial damage, bronchial dilation, impaired clearance of secretions, recurrent infection, and more bronchial damage