A testicular examination is mainly performed on male patients who present with testicular pathology e.g. pain, swelling, a lump. Although titled testicular examination it involves the examination of the penis, scrotum and testes. As this is an intimate examination it is pertinent to gain a good rapport with your patient, maintain good communication and ensure the patient’s dignity at all times. Remember to offer a chaperone for this skill. For the purposes of your exam, you will most likely be examining a mannequin.

A brief screening examination should be conducted checking the face, eyes, mouth, chest, abdomen, spine and limbs to exclude major abnormalities. A strong cry and a widespread pink blush over the face and body are good signs that all is well. Some children may be born with ambiguous genitalia. Ambiguous genitalia is a medical emergency and requires urgent assessment by a paediatrician. If you have sufficient clinical experience, an orogastric tube should be passed when the neonate's mother has suffered polyhydramnios. This excludes oesophageal atresia.

This is how Paraumbilical hernia looks like and how it is examined although it looks very simple but in exam it can be very difficult to perform all steps in small amount of time. This can be short case or even long of #cpsp #fcps #mbbs #medicalstudent #mbbsexams #plab2 #plab #plab1 and MS #genernalknowledge #surgery exams

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Anatomy of The Peritoneal Cavity

Watch that video of Enema Medical Insertion Procedure

In this video, Dr Dhaval Patel, the best brain & spine surgeon in Surat South Gujarat, is performing Brain Hemorrhage Surgery. The Brain Hemorrhage Surgery was successfully done by the best neurosurgeon Dr Dhaval Patel in the midnight in Surat, South Gujarat.

Dr Dhaval Patel is the best and experienced brain & spine surgeon in Adajan, Vesu, Parvat Patiya, Surat, South Gujarat. Dr Dhaval is the expert of treatments and surgery for brain problems and spine problems.
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Brain Hemorrhage Surgery, Best Brain & Spine Surgeon, Neurosurgeon, Brain Tumor Surgery, Brain Treatment Expert, Brain Expert, Brain & Spine Surgery, Neurosurgery in Surat, South Gujarat, Ahmedabad, Rajkot, Anand, Porbandar, patan, kutch, jamnagar, bhavnagar, junagadh, mehsana, nadiad, amreli, morbi, gandhinagar, verval, palanpur,godhra, gandhidham, botad, jetpur, kundal, kalol, disha, gondal, himatnagar, bhuj, modasa, lonavala, mandavi, kheda, khambhaliya, khambhat, dwarka, chhota udaipur, ambaji, dhoraji, idar, vallabhipur, una, dhandhuka, bhachau, mundra.

Dr. Dhaval Patel is an excellent neurosurgeon in Surat, South Gujarat. He is a Brain and Spine Surgeon; he is a reputable Neurosurgeon in Surat, South Gujarat. He has been practicing for the past five years. Till now, he has done 2500+ minor and major surgeries.

NEUROSURGEON DR. DHAVAL PATEL
Specialist in Brain & Spine Surgery
M.S.DNB (Neurosurgery - New Delhi)
Consultant Neurosurgeon

Surat Neuro Clinic Majura Gate, Ring Road, Surat.
Unity Hospital Parvat Patiya, Surat
United Green Hospital Adajan, Surat.
For more info. : +91-9687866766

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Watch that video of full Testicles Autopsy

External cephalic version, or version, is a procedure used to turn a fetus from a breech position or side-lying (transverse) position into a head-down (vertex) position before labor begins. When successful, version makes it possible for you to try a vaginal birth.

To use the diaphragm, first cover the inside of it with spermicide. Then insert it into your vagina so that it covers your cervix. You can insert the diaphragm up to 6 hours before sex. You should leave it in for at least 6 hours after the last time you have sex.

Open heart (coronary artery bypass, or CABG) surgery is performed in order to reroute, or "bypass," blood around blocked arteries, thereby improving the supply of oxygen-rich blood to the heart. Surgeons usually use an artery from the chest wall to construct the "detour" around the blocked part of the artery. Veins from the legs are also used.

Delayed puberty is defined as the absence of any signs suggestive of puberty by 14 years of age. In this case, the patient's pubertal delay appears to be constitutional because of his positive family history, absence of syndromic features or systemic illness, and bone age of 12 years. Puberty correlates more closely with bone age than chronological age. On follow-up, the patient will most likely demonstrate a similar onset of puberty as his father.

A video showing vaginal delivery

Watch that video to know the Female Genital Infections Causes and treatments.

Urinary incontinence isn't a disease, it's a symptom. It can be caused by everyday habits, underlying medical conditions or physical problems. A thorough evaluation by your doctor can help determine what's behind your incontinence. Temporary urinary incontinence Certain drinks, foods and medications can act as diuretics — stimulating your bladder and increasing your volume of urine. They include: Alcohol Caffeine Decaffeinated tea and coffee Carbonated drinks Artificial sweeteners Corn syrup Foods that are high in spice, sugar or acid, especially citrus fruits Heart and blood pressure medications, sedatives, and muscle relaxants Large doses of vitamins B or C Urinary incontinence also may be caused by an easily treatable medical condition, such as: Urinary tract infection. Infections can irritate your bladder, causing you to have strong urges to urinate, and sometimes incontinence. Other signs and symptoms of urinary tract infection include a burning sensation when you urinate and foul-smelling urine. Constipation. The rectum is located near the bladder and shares many of the same nerves. Hard, compacted stool in your rectum causes these nerves to be overactive and increase urinary frequency. Persistent urinary incontinence Urinary incontinence can also be a persistent condition caused by underlying physical problems or changes, including: Pregnancy. Hormonal changes and the increased weight of the uterus can lead to stress incontinence. Childbirth. Vaginal delivery can weaken muscles needed for bladder control and also damage bladder nerves and supportive tissue, leading to a dropped (prolapsed) pelvic floor. With prolapse, the bladder, uterus, rectum or small intestine can get pushed down from the usual position and protrude into the vagina. Such protrusions can be associated with incontinence. Changes with age. Aging of the bladder muscle can decrease the bladder's capacity to store urine. Menopause. After menopause women produce less estrogen, a hormone that helps keep the lining of the bladder and urethra healthy. Deterioration of these tissues can aggravate incontinence. Hysterectomy. In women, the bladder and uterus are supported by many of the same muscles and ligaments. Any surgery that involves a woman's reproductive system, including removal of the uterus, may damage the supporting pelvic floor muscles, which can lead to incontinence. Enlarged prostate. Especially in older men, incontinence often stems from enlargement of the prostate gland, a condition known as benign prostatic hyperplasia. Prostate cancer. In men, stress incontinence or urge incontinence can be associated with untreated prostate cancer. But more often, incontinence is a side effect of treatments for prostate cancer. Obstruction. A tumor anywhere along your urinary tract can block the normal flow of urine, leading to overflow incontinence. Urinary stones — hard, stone-like masses that form in the bladder — sometimes cause urine leakage. Neurological disorders. Multiple sclerosis, Parkinson's disease, stroke, a brain tumor or a spinal injury can interfere with nerve signals involved in bladder control, causing urinary incontinence.

Watch that Ectopic Pregnancy Abortion Surgery

Watch that Male Catheter Insertion Procedure

Watch that video of Tying The Sperm Canal to Avoid Ejaculation

The pathobiology of MM is complex and the root underlying cause of myeloma is the multistep genetic changes in the postgerminal center B cell. In addition, the bone marrow microenvironment plays a crucial role.[2] The interaction between myeloma cells and the microenvironment is mediated through adhesive interactions via cell-surface receptors, paracrine loops involving several cytokines, such as IL-6, VEGF and IL-10, and suppression of cell-mediated immunity.[2–4] IMiDs modulate many of these interactions leading to decreased myeloma cell growth and survival. Thalidomide was the first IMiD introduced to treat MM. It was initially synthesized in Germany in the late 1950s to treat insomnia and morning sickness. It was withdrawn from the market in 1961 because of its teratogenic effects. Its immunomodulatory properties were realized when it was observed to improve erythema nodosum leprosum, a painful immunologic reaction of leprosy, leading to its approval by the FDA in 1998 with tight prescribing and marketing regulations. Subsequent research showed the diverse mechanism of action of thalidomide including its immunomodulatory effect by inhibition of de novo IgM antibody synthesis,[5] modulation of the T-cell subset by increasing the T-helper cells, inhibitory effects on the TNF-α and antiangiogenic activity leading to its use in MM. Significantly higher response rates in combination with dexamethasone led to its approval in the treatment of newly diagnosed MM in 2006. Lenalidomide, a second-generation IMiD, was developed from the structural backbone of the thalidomide molecule by the addition of an amino group (NH2-) at position 4 of the phthaloyl ring and removal of the carbonyl group (C = O) of the 4-amino-substituted phthaloyl ring (Table 1).[6] In addition to immunomodulatory effects, other mechanisms of action have been described such as direct cytotoxicity via induction of apoptosis, inhibition of cell adhesion molecules and inhibition of growth signals that promote bone marrow angiogenesis